Are Spider Silk Gene Sequences in the
C0\/lD \/axxine
Causing Fibrous Blood Clots?
by Jeffrey Dach MD
Sarah, a long term patient in my office doing well on bioidentical hormone replacement told me a story about her cousin who developed blood clots after a C0\/lD booster, requiring abdominal surgery for mesenteric artery occlusion. Thankfully, her cousin is back home recuperating. Since the rollout of the
C0\/lD \/a<<ine, 70% of mortuary embalmers have reported unusual fibrous blood clots in cadavers. (1-2)
Header Image: IV contrast enhanced CAT scan showing Saddle Embolus (Red Arrows blood clot) lodged across bifurcation of pulmonary artery, Transferred from en.wikipedia to Commons. Author Glitzy queen00 at English Wikipedia. public domain. wikimedia commons.
Watch video of Richard Hirschman, Funeral Director And Embalmer Removing Clot From Jugular Vein:
Pathologist Ryan Cole MD, Discusses Unusual Blood Clots which contain amyloid protein: Link
Pathologist, Dr. Ryan Cole, tells Dr. Drew about the strange, foot-long cl0ts he’s been finding in mRNA injected patients—both alive and deceased—and how they can be broken down and cleared from the body using a natural enzyme called ‘Nattokinase’.
“The morticians that started seeing these—when a body comes in and they have to preserve it, they cannulate large vessels, they put their needles in large vessels—they started getting back pressure that they hadn’t experienced before. And there are one or two that have spoken out, but I know of about another 50 that are seeing the same, who want to keep their jobs, so they don’t say anything.”
Biologist Prof. Dr. Ulrike Kammerer, University of Würzburg
Watch this video in which Dr. Ulrike Kammerer says C0\/lD jabs contain spider silk gene sequence, the genetic code for spider silk production (Spidroins which stain for amyloid). Spider silk protein has been the subject of extensive research for the last twenty years and has been successfully produced using recombinant genetic engineering techniques. So, inserting the gene sequence for spider protein is a simple “off the shelf” maneuver to weaponize the C0\/lD \/a<<ine, making it potentially deadly. Thus confirming the nefarious intentions of the creators of this bio-\/\/eap0n. These spider silk fibers could explain the fibrous cl0ts staining for amyl0id found in the vasculature on aut0psies of cadavers who died after C0\/lD jabs. (1-28)
DNA Contamination of mRNA C0\/lD \/a<<ines
A new study from Germany by Dr. Ulrike Kämmerer published in Science, Public Health Policy and the Law December 3, 2024 found high levels of residual DNA in the mRNA
C0\/lD \/a<<ines vials, which far exceeds safety limits.
Decades of gene therapy research has shown DNA contamination causes insertional mutagenesis, and cancer. Dr. Ulrike Kämmerer’s study calls for halting the use of mRNA C0\/lD \/a<<ines until these safety issues are thoroughly investigated and resolved. This study is one of many from a number of independent laboratories calling attention to this flaw in manufacturing the product. About one year ago, Dr. Joseph Ladapo, Surgeon General State of Florida Department of Health has issued a guidance, September 13, 2023, recommending against use of mRNA C0\/lD \/a<<ines because of this DNA contamination.(3-5)
Conclusion: Spider silk protein has been the subject of extensive research for the last twenty years and has been successfully produced using recombinant genetic engineering techniques. So, inserting the gene sequence for spider protein is a simple “off the shelf” maneuver to weaponize the C0\/lD \/a<<ine, making it potentially deadly. Thus confirming the nefarious intentions of the creators of this bio-\/\/eap0n. DNA contamination is a more glaring flaw which should immediately cause the C0\/lD \/a<<ine product to be pulled from the market place, and a halt to the entire program. (1-28)
Articles with Related interest:
Florida Department of Health Advises Against C0\/lD B00STERS
Dr. Karina Whitehouse Presentation on mRNA \/axxines
Jeffrey Dach MD
7450 Griffin Road, Suite 190
Davie, Fl 33314
954-792-4663
References
1) C0\/lD Exclusive: 70% of Embalmers Report Finding Strange Blood Clots Beginning in Mid-2021, Childrens Health Defense by John-Michael Dumais
January 22, 2024
2) Embalmers are Continuing to Find Mysterious Clots in the Vaccinated...Reviewing the results of a recent citizen’s investigation and what we now know about these amyloid clots.
A Midwestern Doctor Feb 22, 2024
3) BioNTech RNA-Based COVID-19 Injections Contain Large Amounts Of Residual DNA Including An SV40 Promoter/Enhancer Sequence
Ulrike Kämmerer Verena Schulz Klaus Steger * Peer Reviewed, Clinical Research
Science, Public Health Policy and the Law 12/03/2024
The study calls for halting the use of mRNA vaccines until these safety issues are thoroughly investigated and resolved.
Conclusion of the study: The findings raise significant safety concerns about the BNT162b2 vaccine due to high levels of residual DNA found in vials, which far exceeds safety limits. The study calls for halting the use of mRNA vaccines until these safety issues are thoroughly investigated and resolved.
4) Just In: Peer-Reviewed German Study Reveals mRNA Vaccine DNA Contamination Exceeds Safety Limits; German Scientists Urge IMMEDIATE HALT!
We call for an immediate halt of all RNA-based biologicals until these concerns are scientifically addressed and convincingly dispelled.
Aussie17 Dec 03, 2024
5) BREAKING: German Study Raises Grave Safety Concerns over BioNTech’s Comirnaty Sonia Elijah Dec 03, 2024
6) Arndt, Tina, et al. “Spidroin N-terminal domain forms amyloid-like fibril based hydrogels and provides a protein immobilization platform.” Nature Communications 13.1 (2022): 4695.
7) Qi, Xingmei, et al. “Spider Silk Protein Forms Amyloid‐Like Nanofibrils through a Non‐Nucleation‐Dependent Polymerization Mechanism.” Small 19.46 (2023): 2304031.
8) Kenney, John M., et al. “Amyloidogenic nature of spider silk.” European journal of biochemistry 269.16 (2002): 4159-4163.
9) Dai, Bin, et al. “Fibril self-assembly of amyloid–spider silk block polypeptides.” Biomacromolecules 20.5 (2019): 2015-2023.
10) Abelein, Axel, et al. “High-yield production of amyloid-β peptide enabled by a customized spider silk domain.” Scientific reports 10.1 (2020): 235.
11) Rising, Anna, et al. “Spider silk proteins: recent advances in recombinant production, structure–function relationships and biomedical applications.” Cellular and Molecular Life Sciences 68 (2011): 169-184.
12)
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Spider Silk Gene Sequence Found In COVID Jabs April 21, 2024
13) Biologist Prof. Dr. Ulrike Kammerer: C0\/lD jabs contain spider silk gene sequence
————————–
14) Spider silk gene sequence found in COVID jabs. GMO spider silk goats, bacteria, worms already exist
15) Scheibel, Thomas. “Spider silks: recombinant synthesis, assembly, spinning, and engineering of synthetic proteins.” Microbial cell factories 3 (2004): 1-10.
16) Abelein, Axel, et al. “High-yield production of amyloid-β peptide enabled by a customized spider silk domain.” Scientific reports 10.1 (2020): 235.
17) Kenney, John M., et al. “Amyloidogenic nature of spider silk.” European journal of biochemistry 269.16 (2002): 4159-4163.
18) Scheibel, Thomas. “Spider silks: recombinant synthesis, assembly, spinning, and engineering of synthetic proteins.” Microbial cell factories 3 (2004): 1-10.
19) Qi, Xingmei, et al. “Spider silk protein forms amyloid‐like nanofibrils through a non‐nucleation‐dependent polymerization mechanism.” Small 19.46 (2023): 2304031.
20) Tjernberg, L. O., et al. “Transmissible amyloid.” Journal of Internal Medicine 280.2 (2016): 153-163.
spidroins are highly soluble and form disordered and partly α-helical structures, but upon passage through the spinning duct, they are converted into solid fibres made up of amyloid-like β-sheets and amorphous parts 48-51. Thus, like amyloid fibrils, silk fibres are formed from soluble proteins under physiological conditions 52, but there are also marked differences between amyloid and silk. Amyloid fibrils are several orders of magnitude smaller and their β-strands are oriented perpendicular to the fibre axis, unlike spider and silkworm silk in which the direction of the β-strands is parallel to the fibre axis 50, 53-55. Furthermore, in contrast to silk proteins, amyloid-forming proteins in their natively folded state generally have specific functions unrelated to fibre formation 56.
21) Kong, Na. “General Methods to Produce and Assemble Recombinant Spider Silk Proteins.” Fibrous Proteins: Design, Synthesis, and Assembly (2021): 57-67.
22) Roth, David Eugene. Genetic Engineering of Functional Large Amyloid Fibers. Diss. Virginia Tech, 2016.
The experimental results show that large amyloid fibers with predictable size and mechanical properties can be anticipated and encoded at the genetic level
23) Pretorius, Etheresia, et al. “Prevalence of amyloid blood clots in COVID-19 plasma.” medrxiv (2020): 2020-07.
We show here that microclots can be detected in the native plasma of COVID-19 patient, and in particular that such clots are amyloid in nature as judged by a standard fluorogenic stain. This provides a rapid and convenient test (P<0.0001), and suggests that the early detection and prevention of such clotting could have an important role in therapy.
24) Exclusive: 70 percent of embalmers report finding strange blood clots beginning in mid-2021
25) Acevedo-Whitehouse, K., and R. Bruno. “Potential health risks of mRNA-based vaccine therapy: a hypothesis.” Medical Hypotheses 171 (2023): 111015.
We propose that in susceptible individuals, cytosolic clearance of nucleotide modified synthetic (nms-mRNAs) is impeded. Sustained presence of nms-mRNA in the cytoplasm deregulates and activates endogenous transposable elements (TEs), causing some of the mRNA copies to be reverse transcribed. The cytosolic accumulation of the nms-mRNA and the reverse transcribed cDNA molecules activates RNA and DNA sensory pathways. Their concurrent activation initiates a synchronized innate response against non-self nucleic acids, prompting type-I interferon and pro-inflammatory cytokine production which, if unregulated, leads to autoinflammatory and autoimmune conditions, while activated TEs increase the risk of insertional mutagenesis of the reverse transcribed molecules, which can disrupt coding regions, enhance the risk of mutations in tumour suppressor genes, and lead to sustained DNA damage. Susceptible individuals would then expectedly have an increased risk of DNA damage, chronic autoinflammation, autoimmunity and cancer. In light of the current mass administration of nms-mRNA vaccines, it is essential and urgent to fully understand the intracellular cascades initiated by cellular uptake of synthetic mRNA and the consequences of these molecular events.
26) Mead, M. Natnaniel, et al. “COVID-19 Modified mRNA “Vaccines” Part 1: Lessons Learned from Clinical Trials, Mass Vaccination, and Censorship by the Bio-Pharmaceutical Complex.” International Journal of Vaccine Theory, Practice, and Research 3.1 (2023): 1112-1178.
27) Mead, M. Nathaniel, et al. “COVID-19 modified mRNA “vaccines”: Lessons learned from clinical trials, mass vaccination, and the bio-pharmaceutical complex, Part 2.” International Journal of Vaccine Theory, Practice, and Research 3.2 (2024): 1275-1344.
28) Gibo, Miki, et al. “Increased Age-Adjusted Cancer Mortality After the Third mRNA-Lipid Nanoparticle Vaccine Dose During the COVID-19 Pandemic in Japan.” Cureus 16.4 (2024).
A recent study showed that SARS-CoV-2 RNA could be reverse-transcribed to DNA and integrated into the human cell genome in vitro [95]. Another study reported that transfected mRNA in the human cells exposed to BNT162b2 leads to unsilencing of the endogenous retrotransposon long interspersed element-1 (LINE-1) and reverse transcription of vaccine mRNA sequences to DNA in the nucleus [96]. Accumulation of vaccine mRNA and reverse-transcribed DNA molecules in the cytoplasm could be expected to induce chronic autoinflammation, autoimmunity, DNA damage, and cancer risk in susceptible individuals [97].
The U.S. Food and Drug Administration (FDA) states in its guidance for the production of viral vaccines for infectious disease, “There are several potential mechanisms by which residual DNA could be oncogenic, including the integration and expression of encoded oncogenes or insertional mutagenesis following DNA integration” [98].
insertional mutagenesis gene therapy
https://www.jci.org/articles/view/35700
Hacein-Bey-Abina, Salima, et al. “Insertional oncogenesis in 4 patients after retrovirus-mediated gene therapy of SCID-X1.” The Journal of clinical investigation 118.9 (2008): 3132-3142.
While 9 of 10 patients were successfully treated, 4 of the 9 developed T cell leukemia 31–68 months after gene therapy. In 2 of these cases, blast cells contained activating vector insertions near the LIM domain–only 2 (LMO2) proto-oncogene.
Jones, Richard J., and Michael R. DeBaun. “Leukemia after gene therapy for sickle cell disease: insertional mutagenesis, busulfan, both, or neither.” Blood, The Journal of the American Society of Hematology 138.11 (2021): 942-947.
Recently, encouraging data provided long-awaited hope for gene therapy as a cure for sickle cell disease (SCD). Nevertheless, the transient suspension of the bluebird bio gene therapy trial (clinicaltrials.gov: NCT02140554) after participants developed acute myeloid leukemia/myelodysplastic syndrome (AML/MDS) raised concerns.
Suspension of myeloablative gene therapy trial
The SCD gene therapy trial HGB-206 (clinicaltrials.gov
: NCT02140554) was suspended between February and June 2021 after 2 participants developed t-MN.8 The first participant developed MDS 3 years after treatment, followed by transformation to AML with monosomy 7; the second participant developed AML 5.5 years after therapy.8 A total of 47 individuals have been treated in bluebird bio SCD gene therapy trials using LentiGlobin: a gene therapy product containing autologous CD34+ cells transduced ex vivo with the BB305 lentiviral vector encoding β-globin, βA-T87Q (clinicaltrials.gov: NCT02140554 and NCT04293185).9
https://onlinelibrary.wiley.com/doi/pdf/10.5402/2012/616310
Romano, Gaetano. “Development of safer gene delivery systems to minimize the risk of insertional mutagenesis‐related malignancies: a critical issue for the field of gene therapy.” International Scholarly Research Notices 2012.1 (2012): 616310.
Integrating gene delivery systems allow for a more stable transgene expression in mammalian cells than the episomal ones.
However, the integration of the shuttle vector within the cellular chromosomal DNA is associated with the risk of insertional
mutagenesis, which, in turn, may cause malignant cell transformation. The use of a retroviral-derived vector system was
responsible for the development of leukemia in five children, who participated in various clinical trials for the treatment of severe combined immunodeficiency (SCID-X1) in France and in the United Kingdom. Unfortunately, the hematological malignancy claimed the life of one patient in 2004, who was enrolled in the French clinical trial. In addition, adeno-associated-viral-(AAV-) mediated gene transfer induced tumors in animal models, whereas the Sleeping Beauty (SB) DNA transposon system was associated with insertional mutagenesis events in cell culture systems. On these grounds, it is necessary to develop safer gene delivery systems for the genetic manipulation of mammalian cells. This paper discusses the latest achievements that have been reported in the field of vector design.
Cesana, Daniela, et al. “A case of T-cell acute lymphoblastic leukemia in retroviral gene therapy for ADA-SCID.” Nature Communications 15.1 (2024): 3662.
Ahmed, Bilal, Maria Zafar, and Muhammad Imran Qadir. “Oncogenic insertional mutagenesis as a consequence of retroviral gene therapy for X-linked severe combined immunodeficiency disease.” Critical Reviews™ in Eukaryotic Gene Expression 29.6 (2019).
Jeffrey Dach MD
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